Worsening Memory May Be Too Quickly Dismissed: Could Leads to Alzheimer's (part 4)

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"Not all subjects with subsequent dementia will experience or report subjective memory impairment at the pre-mild cognitive impairment stage," they conclude.

"However, if subjective memory impairment is present in a subject without cognitive impairment as evidenced by neuropsychological test results, it may inform about the risk for dementia and may contribute to individual decisions about diagnostic procedures and interventions to lower the risk factors for Alzheimer's disease based on current knowledge."

This study was supported by grants from the German Competence Network on Dementia and by the German Competence Network on Degenerative Dementias funded by the German Federal Ministry for Education and Research.

Worsening Memory May Be Too Quickly Dismissed: Could Leads to Alzheimer's (part 3)

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Participants were asked whether they believed their memory was becoming worse and whether or not this caused worry for them (one way for researchers to gauge the severity of memory impairments). They were then followed up one and a half and three years later and tested for mild cognitive impairment and dementia.

Individuals who had memory impairment with concern at the beginning of the study were at the highest risk for conversion to any dementia, or Alzheimer's disease-related dementia, at either follow-up.

"Subjective memory impairment without worry was independently associated with increased risk for dementia," the authors write. "This risk was roughly doubled by the presence of subjective memory impairment-related worry."

In addition, having memory impairment at the beginning of the study and mild cognitive impairment at the first follow-up increased the risk for conversion to any dementia or dementia related to Alzheimer's disease at the second follow-up; these individuals had the greatest risk for developing dementia.

Worsening Memory May Be Too Quickly Dismissed: Could Leads to Alzheimer's (part 2)

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Concept of mild cognitive impairment as a predementia manifestation of Alzheimer's disease is substantiated’

There is more evidence today that the memory problems many have dismissed as just “normal aging,” or have paid little attention to, may be more serious than many believed. This subjective memory impairment or mild deficits in memory appear to predict progression to more advanced stages of cognitive impairment and dementia.

Individuals with cognitive test results below normal ranges but who are still able to participate in most regular activities are said to have mild cognitive impairment, according to background in the report in the April issue of Archives of General Psychiatry, one of the JAMA/Archives journals..

Worsening Memory May Be Too Quickly Dismissed: Could Leads to Alzheimer's (part 2)

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This condition has previously been established as a risk factor for Alzheimer's disease and other forms of dementia, with 10 percent to 20 percent progressing from mild cognitive impairment to dementia each year.

"The concept of mild cognitive impairment as a predementia manifestation of Alzheimer's disease is substantiated by studies providing biologic evidence for the presence of Alzheimer's disease in patients with mild cognitive impairment," the authors write.

"However, Alzheimer's disease-related pathologic changes in the brain evolve several years before the onset of mild cognitive impairment."

Frank Jessen, M.D., University of Bonn, Germany, and colleagues in the German Study on Aging, Cognition and Dementia in Primary Care Patients Study studied 2,415 adults age 75 or older who did not have cognitive impairment at the beginning of the study.

Self test for memory disorders( part 2 )

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Douglas Scharre, a neurologist at the Ohio State University Medical Center, developed the Self-Administered Gerocognitive Examination (SAGE) to help identify individuals with mild thinking and memory impairments at an early stage.

The research shows four out of five people (80%) with mild thinking and memory (cognitive) issues will be detected by this test, and 95% of people who are normal thinking will have normal SAGE scores.

Scharre, who specializes in treating Alzheimer's disease, said treatments for Alzheimer's and dementia are more effective when they are introduced in the earliest stage of the disease. Unfortunately, he said he often sees patients more than three to four years after the first symptoms of a cognitive impairment began to appear.

"It's a recurring problem," said Scharre. "People don't come in early enough for a diagnosis, or families generally resist making the appointment because they don't want confirmation of their worst fears. Whatever the reason, it's unfortunate because the drugs we're using now work better the earlier they are started."

Self-test for memory disorders designed

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Washington, DC: Researchers have designed a self-administered test to screen for early dementia, and it could help speed the diagnosis and subsequent treatment of memory disorders, including Alzheimer's disease.

The handwritten self-assessment, which can take less than 15 minutes to complete, is a reliable tool for evaluating cognitive abilities.

The discovery has been explained in the current issue of the journal Alzheimer Disease and Associated Disorders.

Douglas Scharre, a neurologist at the Ohio State University Medical Center, developed the Self-Administered Gerocognitive Examination (SAGE) to help identify individuals with mild thinking and memory impairments at an early stage.

The research shows four out of five people (80%) with mild thinking and memory (cognitive) issues will be detected by this

Drug may prevent Alzheimer's, slow aging

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UPI

A drug already approved by the U.S. Food & Drug Administration to prevent organ rejection may help prevent Alzheimer's disease, researchers said.

Several weeks after researchers at the University of Texas Health Science Center at San Antonio said the drug rapamycin helped mice engineered to develop Alzheimer's to retain memory, the same researchers said the drug helped an entirely different mouse model engineered to have early Alzheimer's disease to remember how to swim a maze.

"Rapamycin treatment lowered levels of amyloid-beta-42, a major toxic species of molecules in Alzheimer's disease," Veronica Galvan, an assistant professor from the Barshop Institute and the department of physiology at the University of Texas Health Science Center, said in a statement.

"These molecules, which stick to each other, are suspected to play a key role in the early memory failure of Alzheimer's."

The drug is produced by a bacterial product isolated from soil of Easter Island and last year the same researchers and colleagues found rapamycin was shown to extend life in mice.

The current findings are published in the journal PLoS ONE